Cancer Therapy: Preclinical Carbonic Anhydrase IX Promotes Tumor Growth and Necrosis In Vivo and Inhibition Enhances Anti-VEGF Therapy

نویسندگان

  • Alan McIntyre
  • Shalini Patiar
  • Simon Wigfield
  • Ji-liang Li
  • Ioanna Ledaki
  • Helen Turley
  • Russell Leek
  • Cameron Snell
  • Kevin Gatter
  • William S. Sly
  • Richard D. Vaughan-Jones
  • Pawel Swietach
  • Adrian L. Harris
چکیده

Purpose: Bevacizumab, an anti-VEGFA antibody, inhibits the developing vasculature of tumors, but resistance is common. Antiangiogenic therapy induces hypoxia and we observed increased expression of hypoxia-regulated genes, including carbonic anhydrase IX (CAIX), in response to bevacizumab treatment in xenografts. CAIX expression correlates with poor prognosis inmost tumor types andwith worse outcome in bevacizumab-treated patients with metastatic colorectal cancer, malignant astrocytoma, and recurrent

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Carbonic anhydrase IX promotes tumor growth and necrosis in vivo and inhibition enhances anti-VEGF therapy.

PURPOSE Bevacizumab, an anti-VEGFA antibody, inhibits the developing vasculature of tumors, but resistance is common. Antiangiogenic therapy induces hypoxia and we observed increased expression of hypoxia-regulated genes, including carbonic anhydrase IX (CAIX), in response to bevacizumab treatment in xenografts. CAIX expression correlates with poor prognosis in most tumor types and with worse o...

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تاریخ انتشار 2012